In addition, this disease is commonly reported among the female gender besides affecting young adults. At certain incidences, the infection may lead to either physical or cognitive disability (Kalb, 2011). Hence, worsen the ailing person’s condition because it assumes either relapse or progressive state (Kalb 2011).
The disease’s pathophysiology is widely unknown to many, though numerous studies are underway with the intention of ascertaining other factors about it (Lezzoni, 2010). Multiple Sclerosis presents itself in two phases (Lezzoni, 2010). The initial phase encompasses brain hidden damage whereas the second is spinal cord paralysis (Lezzoni, 2010). During these phases, most lesions characterize the silent areas of brain damage thus, becoming difficult for doctors to detect them. What follows is infiltration of immune cells known as demyelination (Lezzoni, 2010). The depiction of this state mostly encompasses white matter and lesions appearing mainly in a peri-ventricular pattern (Lezzoni, 2010). Besides the intense injury of the normal white matter, the illness also damages cortex and profound gray material nuclei (Lezzoni, 2010). Symptoms of this illness majorly occur due to the accumulation of lesions in the central nervous system (Lezzoni, 2010). The most notable symptoms of this disease encompass lapses, which are due to axonal conduction at the trace of the lesions (Lezzoni, 2010).
Multiple sclerosis lesions in the brain and spinal cord lead to the damage of numerous central nervous system parts (Lezzoni, 2010). The disease professes various clinical symptoms that can be either mild or aggressive. For instance, numbness in limbs, slurred speech, fatigue and dizziness which represent mild symptoms, whereas vision impairment, pain in various body parts, lack of body coordination and balance breakdowns encompass aggressive symptoms (Lezzoni, 2010). Infected persons are extremely