Once these ischemic injuries set in, they leave patients with symptoms including high body temperature and other neurological dysfunctions. In such cases, the need to find protective interventions to deal with these aftermath effects becomes highly important and urgent. In this paper, one critical intervention to dealing with post adult cardiac arrest, which has effects like risk of ischemic injury to tissue, which is therapeutic hypothermia discussed for its effectiveness and efficiency as a care approach.
Neurological anatomy and physiology is discussed due to the relationship between neurological wellbeing and post cardiac arrest. As Arrich, Holzer, Havel, Müllner and Herkner (2012) note, “Good neurologic outcome after cardiac arrest is hard to achieve”. An understanding of the neurological anatomy and physiology will therefore make it possible to know why this is so and how this situation can effectively be tackled using therapeutic hypothermia. Quote (year) explained that neurological system comprises of two major components, which are the central nervous system (CNS) and the peripheral nervous system (PNS). Whereas the PNS is mainly made up of nerves that exit from the spinal cord, the CNS comprises the brain, the spinal cord, and the cranial nerves. Three major organs can therefore be anatomically and physiologically identified, which are brain, spinal cord and the vertebral column. Functionally, the brain is responsible for functions including “imagination, memory, speech, and limb movements to secretion hormones and control of various organs within the body” (Sehati, 2009). These functions are however made possible by the collective functioning of other cells, tissues and organs including brain cells, meninges, cerebrospinal fluid (csf), ventricles, brainstem, thalamus, cerebellum, lobes, cerebrum, hypothalamus, pituitary gland, basal ganglia, pineal gland, and cranial