Heart failure increases pressure in the pulmonary circulation, which in turn pushed fluids in the air sacs. Therefore, Murray (2012) observes that pulmonary edema is a condition whereby fluids accumulate in the…
On the other hand, non-cardiogenic pulmonary edema is due to an injury to the lung’s vasculature. Treatment for pulmonary edema is aimed at enhancing the respiratory function, treating the underlying cause, and avoiding more damage to the lung. In severe cases, pulmonary edema can result in respiratory distress, heart attack, and even death. Considering that cardiogenic pulmonary edema (CPE) is a type of pulmonary edema, the paper will focus on cardiogenic pulmonary edema.
Lungs contain numerous small and elastic sacs referred to as alveoli (Luks, 2013). During gaseous exchange, these air sacs take in oxygen and blow out carbon dioxide. In normal cases, the respiration occurs devoid of any problems. However, in certain situations, the alveoli may be filled with fluids in place of air, thus preventing oxygen from being absorbed into the bloodstream. This results in a condition called cardiogenic pulmonary edema (Murray, 2012). Conventionally, cardiogenic refers to the left ventricle causes. In this case, cardiogenic pulmonary edema is as a result of failure of the heart’s left ventricle to effectively eliminate blood from the pulmonary circulation. As noted by Cleland, Yassin, and Khadjooi (2010), this may be attributable to increased pressure in heart leading to a fast or slow heartbeat, and accumulation of fluids.
CPE is caused by problems in the heart muscle, valvular problems, and renal artery stenosis. When the mean pulmonary pressure increases from the normal of 15 mmHg to over 25 mmHg or is ...
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According to the American Association Guidelines, the procedure should only be done if the patient remains in a coma state following the return of spontaneous circulation. Numerous studies have been done to identify the risks and benefits associated with this procedure.
al., 2008; GOLD, 2008; National Lung Heart and Heart Blood Institute, 2009). COPD is a clinical syndrome that results to excessive production of mucous, wheezing, continual dyspnea, and chest tightness associated with progressive obstruction of expiratory airflow secondary to persistent inflammation of the airways and lung (Woodley and Whelan, 1992; Sutherland and Cherniack, 2004; Fabbri, et al., 2008).
CHF due to diseased heart: mitral regurgitation, mitral stenosis, myocardial infarction, etc. In high output failure heart is normal, but metabolic need of body is drastically increased and heart is not able to fulfill the required need, e.g. severe anemia, thyrotoxicosis, etc.
Due to reduced elasticity and a larger fibrous barrier, diffusion of oxygen and carbon dioxide between the alveoli and blood stream is hindered. This hindrance causes low lung compliance and lowers the effective rate of gaseous diffusion between alveoli and blood stream.
Reports bared that there are already millions of people affected by COPD, though not transmissible but some of them are infected with this disease without medical knowledge on it and thus bereft with knowledge on how to go about with this disease for medication.
Pulmonary edema is the name of a condition that is caused as a result of excessive accumulation of fluid inside the lungs. Lungs inhale oxygen and the carbon dioxide is passed for exhalation into the alveoli.
Clinical Pharmacology: Protein Tyrosine Kinase Inhibitor, inhibits BCR-ABL tyrosine kinase, half life of 18 hours, 40 hours for the metabolite, peak plasma time is 2-4 hours with 98% bioavailability, 95% protein bound, metabolised by CYP3A4; clearance is
Basically, there are two causes of pulmonary edema. The first is the inability of the heart’s left ventricle to be able to remove the blood completely from the pulmonary circulation. The second reason is usually cited as
At the cellular level, the malformation is associated with abnormalities of chromosomes with trisomy 21, 18 and 13. Tetralogy of the fallot can also be associated with chromosome 22 and microdeletions. There is a 3 percent risk of recurrence of in families. Some untreated
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