The positive association between development of central obesity and increased cortisol levels has been demonstrated by numerous studies (Dimitriou, Maser-Gluth & Remer, 2003; Livingstone et al, 2000; Tiosano et al, 2003). Increased cortisol secretion results in an increased energy intake in both males and females (Epel et al, 2001). The study by Gluck et al (2004) and Gluck (2006) showed that increased cortisol secretion, a major component of the stress response, could play an important role in the pathogenesis of binge eating, leading to the development of obesity by increasing food intake. Increased basal cortisol levels have also been found in bulimia nervosa contributing to a great amount of hunger and binge eating. An increased cortisol level has been shown to be positively related to central fat distribution in both obese and lean individuals (Bjorntorp, et al, 2000; Douyon et al, 2002). The study by Epel et al (2000) supports the hypothesis that stress-induced cortisol secretion may contribute to central fat and demonstrate a link between psychological stress and risk for disease in women. This relationship holds true for men as well. Besides obesity, increased cortisol levels are also associated with, a profound alteration of intermediary metabolism, insulin resistance, changes in lipid metabolism, hypertension and cardiovascular abnormalities. In these obese subjects, levels of cortisol in plasma, saliva or urine may be significantly higher than those of matched normal subjects. (Andrew et al, 1998
This research paper “Does stress-induced cortisol contribute to central obesity?" is based on an observation within writers clinic where patients have central obesity and are often stressed. Moreover, is an analysis of how diet, relaxation, and exercise can have positive effects…
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